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Similarly, hearts from transgenic mice overexpressing GPx1 had preserved mitochondrial and cardiac function in the presence of doxorubicin, with increased efficiency of mitochondrial respiration and better contractility and diastolic function compared wild type hearts exposed to doxorubicin [44]

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411 G i2 also interacts with the engulfment and cell motility 1/dedicator of cytokinesis 1 (Elmo1/Dock180) complex, which acts as a RacGEF and activates Rac proteins, inducing actin polymerization and cell migration