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Interestingly, it was previously proposed that the ability of KEAP1 to sense H 2 O 2 depends on the formation of a disulfide bond between Cys 226 and 613, and this sensing mechanism differs from that used by electrophilic NRF2 inducers, suggesting the plausibility of cysteinylation of these two residues in cells [25]

Heart Rhythm, 14 (12), 18491855

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"Improvement of alveolar glutathione and lung function but not oxidative state in cystic fibrosis." Am J Respir Crit Care Med

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Growth deficiency may be less critical for understanding the disabilities of FASD than the neurobehavioral sequelae to the brain damage