Hepatic steatosis is a consequence of lipid acquisition exceeding lipid disposal, i.e., the uptake of fatty acids and de novo lipogenesis surpassing fatty acid oxidation and export
Peptides are gaining traction for several key reasons: Specificity and targeting Unlike some medications that have broad, systemic effects (and consequently, systemic side effects), peptides are highly specific
A multiomics framework that integrates transcriptomic, proteomic, and metabolomic analyses, coupled with CRISPR-based functional validation, will be essential to clarify these hierarchical relationships and decode the broader -CCresponsive network across plant systems
Two practical consequences fall out of this mechanism, and both explain the shape of the protocol below
* Correspondence: Pablo Albertos, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
doi: 10.1002/jsfa.6179 47 HuntJR
